2 Gluten free raw bread alternatives, the sweet and the savory


The summer has been great, the parties have been fun, my good eating habits sort of crashed.  Now its time to get back into feeding my body without the unnecessary crap.....sugar, gluten, and eliminate some dairy products.  These foods keep me from feeling my best. Although some of the lyme symptoms can flair, eating right, for me, can be one of my best defences. 
Here is how I am getting back on the wagon, starting up the dehydrator again, on the rainy days. 

Busy today,  with no sun and cool weather it's time to turn on the dehydrator. Its a good day for some savory raw crackers, and a sweet raw banana bread....No sugar,  no gluten, no dairy. Just homemade goodness and organic ingredients bring out the best in yourself and feel the love as the good nutrition is absorbed, feed the body, feed the brain, detox.

Beet Carrot Ginger Curcumin Flax Crackers with Nutritional Yeast and Sea Salt.


the beets make a beautiful color, I spread them thin for a faster drying time.  Try to fill up the dehydrator as much as possible in order to save electricity.  The raw banana bread is also in.
Here are the finished crackers with my fermented salsa and a cup of kombucha, mmmm.

                                                                                      

Raw Banana Bread 
 Love this! dehydrate the small loaves over night 8-12 hours depending on how dry or soft you like it........ with flax, dates, raw honey, 4-7 bananas, cinnamon, vanilla, nutmeg, psyllium husk (water if needed). Tomorrows breakfast.   This can be frozen for future use. Going in....
                and finished.....
a little homemade plum jam will top it off, oh ya!
                  


5-MTHF supplementation, Folate metabolism- getting your energy back

The Folic Acid Story





methyl-B12, trimethylglycine (TMG), or SAMe may help restore normal ranges (11).

B12, SAMe, TMG
The end product of folate metabolism, 5-MTHF, is an essential precursor for neurotransmitter metabolism and the synthesis of dopamine, serotonin, and norepinephrine. It is also necessary for the production of SAMe, which participates in over 200 enzymatic reactions in the body.

Did the FDA’s attempt to reduce birth defects have unintended consequences? In the 1990’s, the US and UK mandated the enrichment of all grain products with folic acid in hopes of reducing the occurrence of neural tube defects. 

In the decades since neural tube defects have indeed declined yet little long term research has been done to determine the effects excess folate might have on non-pregnant adults. The tolerable upper limit (TUL) for folate is set at 1,000 mcg/day for adults, but with the overconsumption of fortified grain products many may exceed this threshold. 

Recent studies are now taking a closer look at potential health risks of folic acid fortification for adults. Folate is not only a concern for adults, but may have particular relevance for some people with Chronic Fatigue Syndrome (ME/CFS).

What Role Might folate play in ME/CFS? folate is a vital component of the single carbon pool that participates in basic cellular processes such as amino acid metabolism, synthesis of nucleotides (DNA, RNA, ATP), and methylation (including epigenetic methylation of DNA). Low levels of folate are implicated in increased risk of cardiovascular disease, stroke, dementia, depression, cancers, and birth defects. Similarly, high levels are also associated with carcinogenesis (1). Folate metabolism is a complex, multi-step process that occurs in every cell in the body. It requires three key enzymes: dihydrofolate reductase (DHF), methylenetetrahydrofolate reductase (MTHFR), and methionine synthase (MTR). 

When you consume the synthetic form of folate – folic acid – in enriched foods or supplements, it must be converted to different forms utilizing these enzymes. If this conversion is inadequate, functional folic acid deficiencies can emerge that may dampen methylation, amino acid metabolism, etc. Despite the fact that this biochemistry has been known for decades, few studies have examined the exact mechanisms by which folic acid is taken up or metabolized in the body after consumption. Those studies which have examined folic acid metabolism have relied only on mouse models which have substantially different concentrations of the above enzymes than humans (2). 

A recent study in patients with stable liver disease indicated, however, that synthetic folic acid from traditional supplements was inefficiently absorbed in the intestinal tract compared to the active 5-methyl-hydrofolate (5-MTHF) form (which was taken up twice as readily).  The researchers concluded that food regulatory bodies should rethink their position on folic acid enrichment. 

Interestingly, the FDA and European Food Standard Agency have already approved products containing 5-MTHF derivatives to replace folic acid (3).

A Key Metabolic Factor – More Biochemistry 
B12, an essential co-factor in folic acid metabolism, is often low in ME/CFS The folate cycle feeds into other key biochemical cycles as well. The end product of folate metabolism, 5-MTHF, is an essential precursor for neurotransmitter metabolism and the synthesis of dopamine, serotonin, and norepinephrine. It is also necessary for the production of SAMe, which participates in over 200 enzymatic reactions in the body.

After 5-MTHF is produced, it is then recycled back into the cycle through the enzyme methionine synthase (MTR). This reaction requires adequate B12 as a cofactor. With B12 deficiency in near epidemic proportions in the elderly, vegetarians, chronic antacid users, SIBO, and people with ME/CFS or FM, it’s possible that folic acid can become trapped as 5-MTHF in what is called the “folate trap.” This “folate trap” prevents the recycling of 5-MTHF back to THF and slows methylation and many other downstream reactions. 

Finally, there is a growing understanding of the link between hypothyroidism and proper folate cycle function. Many ME/CFS and FM patients have subclinical hypothyroidism that may be related to inadequate production of 5-MTHF. This methylated form of folate is essential for the conversion of tyrosine to thyroid hormone (1). 

Who With ME/CFS or Fibromyalgia Should Be Concerned With This Study? It’s possible but not certain that people with ME/CFS and FM may already be behind the eight ball, so to speak, regarding folate metabolism. Dr. Amy Yasko and the late Rich VonKoynenberg proposed that genetic mutations (single nucleotide polymorphisms or SNPs) in enzymes involved in the folate methylation pathway may be common in the ME/CFS and/or FM populations. Of the three enzymes involved in folate metabolism, methylenetetrahydrofolate reductase (MTHFR) plays the largest role in the conversion and utilization of folates. A genetic defect in this key enzyme could dramatically affect the ability to convert, recycle, and utilize folates. 

Genetic Aspects An Open Medicine Institute study is examining the effects of MTHFR mutations on folate metabolism in ME/CFS. Two common genetic variations, C677T and A1298C, have been identified in the MTHFR gene that codes for the MTHFR enzyme. The C677T type has been associated with increased incidence of cardiovascular disease and other health risks (4). This MTHFR SNP has been found to be prevalent in certain populations of people. Heterozygous MTHFR C677T, meaning one copy of the gene is mutated while the other is normal, is found in 42% of Hispanics, 35% of Caucasians, and 14% of African Americans. Homozygotes, where both copies of the gene are mutated, are less common in the general population with 10% preponderance (5).

Further research and stricter study design are certainly warranted. Another study measuring levels of homocysteine in the cerebrospinal fluid of CFS/FM patients found elevations suggestive of problems recycling folate back to methionine. However, it was unclear whether these findings were due to genetic SNPs in enzymes, folate deficiencies, or B12 deficiencies (8). 

The Open Medicine Institute is currently engaged in a study to determine the extent of MTHFR mutations and the role they may play in aberrant folate metabolism in ME/CFS. 

What Problems Might High Levels of Unmetabolized Folic Acid (UMFA) Pose?

If enzymatic road blocks from genetic mutations or inadequate enzyme cofactors inhibit synthetic folic acid metabolism, high concentrations of folic acid can spill over into the portal circulation. A natural killer connection, of all things, may be possible Elevated levels of unmetabolized folic acid (UMFA) have been implicated in increased overall mortality (9).  Epidemiological studies have found a striking association between the incidence of colon cancers and folic acid food enrichment. In the US, rates of colorectal cancer began to increase in 1996 and reached a maximum in 1998. Rates have continued to exceed those present during folic acid pre-enrichment days by as much as 15,000 extra cases per year (2).

 Worth mentioning is that these observational studies are inadequate to determine causation due to the inability to control for all possible confounders. Nonetheless, hindsight tells us that careful study of genetic and biochemical factors should have been more thoroughly investigated before implementing a potentially harmful change in food policy. 

Some studies also speculate that excess folic acid may be related to decreased natural killer cell cytotoxicity (10). This could be due to the fact that excess naturally occurring folate (DHF) blocks normal function of the MTHFR enzyme providing another potential block in methylation and other downstream biochemical cycles. 

More study is urgently needed to determine if synthetic folates hinder immune functioning. 

Recommendations for People Who Are Concerned? CFS/FM patients and perhaps everyone should check their methylation status utilizing genetic testing such as 23andMe (see www.23andme.com for $99).  Conventional laboratories like Quest or LabCorp also now have MTHFR studies that may be ordered by a healthcare provider. Specialty labs like SpectraCell or HealthDiagnostics specialize in more in-depth methylation studies. (The FDA now allows 23andme to only provide raw data.   Several free online tools can, however, do that job. Yasko has an excellent tool plus there’s http://geneticgenie.org/and http://www.promethease.com)

Dietary and Supplementation Suggestions

If MTHFR SNPs are found, consider avoiding all forms of synthetic folate and folic acid—including those found in grain products. (Regardless, avoiding grains due to the high incidence of gluten sensitivity in CFS and FM patients is sometimes recommended.)
Some supplement changes may be in order 
Foods high in naturally occurring folate (dihydrofolate, DHF)
spinach, turnip greens, beans, sunflower seeds, Brewer’s yeast, liver.
Foods often fortified with folic acid breakfast cereal, bread, flour, corn meal, pasta, rice. When choosing supplements, opt for a multi-vitamin or B-complex with active forms of folate such as folinic acid or 5-MTHF

To assess if supplementation is sufficient to bypass methylation defects, routine homocysteine, folate, and B12 can be measured. Folate is most accurately measured as red blood cell folate. Serum B12 measurements are notoriously inaccurate, so methylmalonic acid (MMA) is the preferred test to perform. If MMA is depressed and homocysteine remains high, there may be an additional need for methyl donors to recycle folate through the cycle. Supplemental methyl-B12, trimethylglycine (TMG), or SAMe may help restore normal ranges (11).

Read more: The Folic Acid Controversy, MTHFR and Chronic Fatigue Syndrome http://www.cortjohnson.org/blog/2014/08/12/folic-acid-controversy-mthfr-chronic-fatigue-syndrome/

1  Kelly GS. Folates: Supplemental Forms and Therapeutic Applications. Alt Med Rev 1998. 3(3): p. 208-220. http://www.ncbi.nlm.nih.gov/pubmed/9630738 2  Smith AD, Kim YI, Refsum H. Is folic acid good for everyone? Am J Clin Nutr March 2008 vol. 87 no. 3 517-533. http://www.ncbi.nlm.nih.gov/pubmed/18326588 3  Patanwala I, et al. Folic acid handling by the human gut: implications for food fortification and supplementation. Am J Clin Nutr. 2014 Jun 18;100(2):593-599. [Epub ahead of print]. http://www.ncbi.nlm.nih.gov/pubmed/24944062 4  Klerk M, et al. MTHFR 677C⟶T Polymorphism and Risk of Coronary Heart Disease: A Meta-analysis. JAMA 2002;288:2023-31. http://www.ncbi.nlm.nih.gov/pubmed/12387655 5  Hughes LB et al. Racial or ethnic differences in allele frequencies of single-nucleotide polymorphisms in the methylenetetrahydrofolate reductase gene and their influence on response to methotrexate in rheumatoid arthritis. Ann Rhuem Dis 2006; 65: 1213-1218. http://www.ncbi.nlm.nih.gov/pubmed/16439441 6  Botto Lorenzo, Yang Quanhe. 5,10-Methylenetetrahydrofolate Reductase Gene Variants and Congenital Anomalies: A HuGE Review. Am. J. Epidemiol. 2000; 151(9): 862-877. http://www.ncbi.nlm.nih.gov/pubmed/10791559 7  Regland,B, Andersson M, Abrahamsson L, Bagby J, Dyrehag LE, Gottfries CG. (1997) Increased concentrations of homocysteine in the cerebrospinal fluid in patients with fibromyalgia and chronic fatigue syndrome. Scand J Rheumatol. 26(4):301-7. http://www.ncbi.nlm.nih.gov/pubmed/9310111 8  Harmon DL, McMaster D, McCluskey DR, Shields D, Whitehead AS. (1997) A common genetic variant affecting folate metabolism is not over-represented in chronic fatigue syndrome. Ann Clin Biochem. 34 (Pt 4):427-9. http://www.ncbi.nlm.nih.gov/pubmed/9247678 9  Neuhouser ML, et al. Mathematical modeling predicts the effect of folate deficiency and excess on cancer-related biomarkers. Cancer Epidemiol Biomarkers Prev. 2011 Sep;20(9):1912-7. Epub 2011 Jul 13. http://www.ncbi.nlm.nih.gov/pubmed/21752986 10  Troen AM, et al. Unmetabolized folic acid in plasma is associated with reduced natural killer cell cytotoxicity among postmenopausal women. J Nutr. 2006 Jan;136(1):189-94. http://www.ncbi.nlm.nih.gov/pubmed/16365081 11  Olthof Margreet, Trinette van Vliet, Esther Boelsma, Petra Verhoef. Low Dose Betaine Supplementation Leads to Immediate and Long Term Lowering of Plasma Homocysteine in Healthy Men and Women. J. Nutr. 2003 Dec. 1; 133(12): 4135-4138. http://www.ncbi.nlm.nih.gov/pubmed/14652361  

Kombucha Benefits, Evidence in the Studies

                                        kombucha scooby available at healthyeats.nl

Fermented tea known as kombucha has been rapidly growing in popularity among health food lovers.  But it's actually been around for thousands of years.  It originated in China in the third century B.C.  Then it made its way to Russia, India, and Japan where it became a staple among Samurai warriors.  Today it is also popular in Poland, Germany, Bulgaria, Indonesia and many other cultures. 
Kombucha – or "the booch" to its fans – is made from green, black or white tea.  It's fermented for at least a week with sugar and a fungal culture consisting of a mixture of bacteria and yeast.  The starter is called a "SCOBY," which is an acronym for "symbiotic colony of bacteria and yeast."
The culture, also sometimes referred to as the "mother," resembles a light brown, tough, gelatinous disk, which is a living, growing organism. With each batch of the tea, the organism can regenerate and create a new culture called the "baby," which can be shared with a friend much like the sharing of a sour dough starter.
Sometimes called "mushroom tea," kombucha has been associated with a long list of health benefits.  It's a probiotic drink with helpful bacteria that support digestion and the immune system. It also contains enzymes, amino acids, antioxidants and polyphenols.
But relatively little scientific evidence has confirmed the health claims of traditional cultures drinking the tea.  Recently, researchers from the University of Latvia gathered 75 studies attesting to the proven health properties of kombucha.[1]
Here are 18 healthy reasons they suggest to raise a glass of Kombucha

1.  Detoxification
Kombucha contains substantial amounts of glucuronic acid (GA).  GA is well known as a detoxicant.  In the body it combines with toxins like pharmaceuticals and environmental pollutants.  It then converts them into compounds that are soluble and the body can excrete.   Drinking kombucha may also help prevent tissues from absorbing industrial toxins in the environment. 

2. Antioxidants
Kombucha  contains abundant antioxidants including vitamins E, C, beta-carotene, and other carotenoids.  Like black tea, Kombucha  also contains polyphenols and other compounds with antioxidant powers.  But because it is fermented, Kombucha is much more powerful than plain tea.  Its antioxidant activity has been found to be 100 times higher than vitamin C and 25 times higher than vitamin E.[2]  For that reason drinking traditional Kombucha may help cure chronic illnesses caused by oxidative stress. 

3.  Energy
Kombucha sets iron free from black tea.  That helps increase levels of blood hemoglobin, and improves oxygen flow to tissues.   It also improves the body's absorption of other non-heme (plant-derived) iron. 

4.  Immunity
Oxidative stress suppresses the immune system, kombucha's high levels of vitamin C support immunity.  Its antioxidant power protects against cell damage, inflammatory diseases, suppressed immunity, and tumors.

5.  Gastric Illnesses
Nonsteroidal anti-inflammatory drugs (NSAIDs) are toxic to the gut.  They can lead to gastric ulcers.Indomethacin, a popular NSAID, can disrupt blood circulation to the stomach's mucous membrane.  Kombucha has been shown to effectively heal gastric ulceration.  Researchers believe the fermented tea protects the mucin content of the stomach.  Its antioxidant activity also protects the lining of the gut.  Kombucha also reduces gastric acid secretion that can damage the mucous membrane.  In fact, kombucha has been found as effective in healing ulcers as prescription omeprazole (brand name Prilosec).[3]

6.  Obesity
Kombucha helps balance the metabolism.  Animal studies show the tea may cause weight loss by encouraging calorie restriction.

7.  Diabetes
Research dating back to 1929 found kombucha can decrease blood sugar levels.  More recent animal studies report that kombucha significantly reduces blood sugar levels in diabetic rats.[4]   Another study suggested kombucha may be considered a candidate for the treatment and prevention of diabetes.[5]

8.  Kidney Toxicity
Kombucha may help eliminate kidney damage caused by environmental pollutants and may be beneficial to patients suffering from renal impairment.[6] Kombucha has also been used to prevent calcification in the kidney and may prevent the formation of kidney stones.[7]

9.  Endothelial Function
Oxidative stress can damage the lining of blood vessels.  That damage is a precursor to atherosclerosis, and a threat to heart health.  Antioxidants in Kombucha help promote regeneration of cellular walls in blood vessels.[8]

10.  Atherosclerosis
In clinical trials involving 52 atherosclerotic patients with high cholesterol, Kombucha helped lower levels to normal. In studies involving ducks, kombucha significantly reduced levels of LDL cholesterol and simultaneously raised HDL levels after just 10 days.  Other animal studies show Kombucha may decrease total cholesterol as much as 45–52%.  It may also significantly decrease triglyceride and LDL levels while increasing HDL.[9]

11.  Hypertension
Kombucha has been used to prevent headaches and dizziness caused by hypertension.  It's been recommended for treating high blood pressure.

12.  Anemia
Organic acids found in Kombucha convert trivalent iron compounds from plant sources to divalent iron ions.  This makes iron from plant sources more available to the body.  And vitamin C in Kombucha enhances iron absorption.   Researchers suggest Kombucha is particularly recommended for elderly people and vegetarians because it enhances the absorption of iron and helps prevent iron deficiency.

13.  Liver Function
Kombucha protects against liver toxicity in animals from overdoses of acetaminophen (Tylenol).[10]  Researchers suggest it might provide a useful therapy for humans as well.[11]

14.  Nervous System
Kombucha contains several amino acids, methylxanthine alkaloids (caffeine, theophylline, and theobromine), ascorbic acid (vitamin C), and B vitamins (including folic acid-B9), necessary for normal metabolism in the nervous system.  It can help with headaches, nervousness, and epilepsy prevention.  It may also prevent depression in the elderly.

15.  Asthma
Daily Kombucha may help asthma patients.  It contains significant amounts of theophyline, a bronchodilator.[12]  The treatment dose of theophyline is 0.18–1.0 g daily.  Just one cup of kombucha contains about 1.44 mg.

16.  Joint Problems
Glucuronic acid inKombucha can be converted by the body into glucosamine, chondroitin-sulphate, and other polysaccharides and glucoproteins associated with cartilage, collagen, and the fluid that lubricates joints.[13] It may also help relieve arthritis, rheumatism, and gout.   

17.  Cancers
Consumption of Kombucha has been associated with lower cancer rates.  Researchers believe it increases the immune system's anticancer defenses.   It may prevent cancer proliferation at early stages of tumor growth due to its glucuronic, lactic, and acetic acid content, as well as its antibiotic compounds.  It may have anticarcinogenic effects especially for hormone-dependent tumors.
Cell studies suggest it may be useful for prostate cancer treatment and prevention.[14]  It's also been studied as an anticancer agent against human lung, osteosarcoma, and renal cancer cell lines.

18.  Antibiotic Resistant Infections
Kombucha contains strong antibacterials to combat infectious diseases such as diptheria, scarlet fever, influenza, typhoid, paratyphoid fever, and dysentery.[15]  Its high total acidity makes it effective against Helicobacter pyloriSalmonella typhimuriumStaphylococcus aureus, and Bacillus cereus.  It's been suggested that kombucha may be an effective alternative to synthetic antimicrobials that are becoming increasingly ineffective.[16]  

The Latvian researchers note that a series of microbiological and biochemical tests on kombucha have repeatedly found it safe for human consumption.  Sally Fallon, author of Nourishing Traditions: The Cookbook that Challenges Politically Correct Nutrition and the Diet Dictocrats, quotes an FDA official as saying that when reasonable care is taken, "you're more likely to find contamination in a cup of coffee than in a cup of properly prepared kombucha."
But there have been isolated reports of sickness after drinking kombucha.  Alternative health guru Dr. Andrew Weil does not recommend drinking the homemade version for fear of contamination with aspergillus, a toxin-producing yeast which he believes would be risky for those with already compromised immune systems, like AIDS patients and cancer patients, as well as for pregnant and nursing mothers.

What to do? If you are healthy, try the commercial brands for yourself. For beginners, a good brand to try is GT's Organic Raw Kombucha.  The founder claims he began making kombucha in 1995 after his mother successfully used the drink in her battle against breast cancer.

It's best to use Kombucha in moderation to begin (four to eight ounces a day), even though the bottle may recommend a full 16 ounces per day. And at almost four dollars a bottle, many people may want to keep their intake moderate.
Another reason to start slowly is that Kombucha has a detoxifying effect.  If the liver is not functioning properly, it can be overwhelmed by the toxins being released. This may be why some people report an allergic reaction to the tea. For that reason, some experts recommend drinking plenty of water while taking kombucha to help flush toxins from the body.

If you are tempted to make your own kombucha, educate yourself first and follow instructions carefully, including using clean equipment, correct temperatures and glass containers.
Kombucha is not a drug but a traditional drink with many health-promoting properties. As with all foods, moderation is key.  It's important to listen to your own body to determine whether it is a good choice for you.

Abstract Title:
Kombucha fermentation and its antimicrobial activity.
Abstract Source:

J Agric Food Chem. 2000 Jun;48(6):2589-94. PMID: 10888589
Abstract Author(s):
G Sreeramulu, Y Zhu, W Knol
Abstract:

Kombucha was prepared in a tea broth (0.5% w/v) supplemented with sucrose (10% w/v) by using a commercially available starter culture. The pH decreased steadily from 5 to 2.5 during the fermentation while the weight of the "tea fungus" and the OD of the tea broth increased through 4 days of the fermentation and remained fairly constant thereafter. The counts of acetic acid-producing bacteria and yeasts in the broth increased up to 4 days of fermentation and decreased afterward. The antimicrobial activity of Kombucha was investigated against a number of pathogenic microorganisms. Staphylococcus aureus, Shigella sonnei, Escherichia coli, Aeromonas hydrophila, Yersinia enterolitica, Pseudomonas aeruginosa, Enterobacter cloacae, Staphylococcus epidermis, Campylobacter jejuni, Salmonella enteritidis, Salmonella typhimurium, Bacillus cereus, Helicobacterpylori, and Listeria monocytogenes were found to be sensitive to Kombucha. According to the literature on Kombucha, acetic acid is considered to be responsible for the inhibitory effect toward a number of microbes tested, and this is also valid in the present study. However, in this study, Kombucha proved to exert antimicrobial activities against E. coli, Sh. sonnei, Sal. typhimurium, Sal. enteritidis, and Cm. jejuni, even at neutral pH and after thermal denaturation. This finding suggests the presence of antimicrobial compounds other than acetic acid and large proteins in Kombucha.
Study Type : In Vitro Study
Additional Links
Pharmacological Actions : Anti-Bacterial Agents : CK(978) : AC(321)

View the Evidence: 

Pubmed Data : Food Funct. 2010 Dec ;1(3):284-93. Epub 2010 Nov 3. PMID: 21776478
Study Type : Animal Study

Article Publish Status : This is a free article. Click here to read the complete article.
Pubmed Data : BMC Complement Altern Med. 2012 ;12:63. Epub 2012 May 16. PMID: 22591682
Study Type : Animal Study

Pubmed Data : J Ethnopharmacol. 2000 Jul;71(1-2):235-40. PMID: 10904168
Study Type : Animal Study

Pubmed Data : Biol Trace Elem Res. 2002 Nov;89(2):105-10. PMID: 14631833
Study Type : Animal Study

Pubmed Data : Biomed Environ Sci. 2001 Sep;14(3):207-13. PMID: 11723720
Study Type : Animal Study

Pubmed Data : J Microbiol Biotechnol. 2009 Apr;19(4):397-402. PMID: 19420997
Study Type : Animal Study

Pubmed Data : J Environ Biol. 2010 Sep;31(5):615-21. PMID: 21387911
Study Type : Animal Study

Pubmed Data : J Agric Food Chem. 2000 Jun;48(6):2589-94. PMID: 10888589
Study Type : In Vitro Study

Pubmed Data : J BUON. 2008 Jul-Sep;13(3):395-401. PMID: 18979556
Study Type : In Vitro Study

Pubmed Data : Indian J Exp Biol. 2011 Jul ;49(7):511-24. PMID: 21800502
Study Type : In Vitro Study


References
http://www.greenmedinfo.com/Margie King is a graduate of the Institute for Integrative Nutrition®
[1] Ilmara Vina et al, "Current Evidence on Physiological Activity and Expected Health Effects of Kombucha Fermented Beverage." J Med Food 17 (2) 2014, 179–188 DOI: 10.1089/jmf.2013.0031
[2] Adriani L, Mayasari N, Kartasudjana RA: "The effect of feeding fermented kombucha tea on HLD, LDL and total cholesterol levels in the duck bloods." Biotechnol Anim Husb 2011;27:1749–1755.
[3] Banerjee D, Hassarajani SA, Maity B, Narayan G, Bandyopadhyay SK, Chattopadhyay S: Comparative healing property of kombucha tea and black tea against indomethacin-induced gastric ulceration in mice: possible mechanism of action. Food Funct 2010;1:284–293.
[4] Shenoy C: "Hypoglycemic activity of bio-tea in mice." Indian J Exp Biol 2000;38:278–279.
[5] Aloulou A, Hamden K, Elloumi D, Ali MB, Hargafi K, Jaouadi B, Ayadi F, Elfeki A, Ammar E: Hypoglycemic and antilipidemic properties of kombucha tea in alloxan-induced diabetic rats. BMC Complement Altern Med 2012;12:63.
[6] Gharib OA: "Effects of kombucha on oxidative stress induced nephrotoxicity in rats." Chin Med2009;4:23.
[7] Dufresne C, Farnworth E: "Tea, kombucha and health: a review."  Food Res Int 2000;33:409–421.
[8] Dufresne C, Farnworth E: "Tea, kombucha and health: a review."  Food Res Int 2000;33:409–421.
[9] Suhartatik N, Karyantina M, Marsono Y, Rahayu ES, Kuswanto KR: "Kombucha as anti hypercholesterolemic agent (in vitro study using SD rats)." Proceedings of the 3rd International Conference of Indonesian Society for Lactic Acid Bacteria (3rd IC- ISLAB): Better Life with Lactic Acid Bacteria: Exploring Novel Functions of Lactic Acid Bacteria, 2011, Yogyakarta, Indonesia.
[10] Pauline T, Dipti P, Anju B, Kavimani S, Sharma SK, Kain AK, Sarada SKS, Sai Ram M, Ilavazhagan G, Kumar D, Selvamurthy W: "Studies on toxicity; anti-stress and hepatoprotective properties of kombucha tea." Biomed Environ Sci 2001;14:207–213.
[11] Jalil A, Amin D, Mohammad HF, Saeid H: "Protective effect of kombucha tea against acetaminophen-induced hepatotoxicity in mice: a biochemical and histopathological study." Comp Clin Path 2012;21:1243–1248.
[12] Pasha C, Reddy G: "Nutritional and medicinal improvement of black tea by yeast fermentation."Food Chem 2005;89:449–453.
[13] Jayabalan R, Marimuthu S, Swaminathan K: "Changes in content of organic acids and tea polyphenols during kombucha tea fermentation." Food Chem 2007;102:392–398.
[14] Sriharia T, Arunkumarb R, Arunakaranb J, Satyanarayanac U: "Downregulation of signalling molecules involved in angiogenesis of prostate cancer cell line (PC-3) by kombucha (lyophilized)."Biomed Prev Nutr 2012;3:53–58.
[15] Talawat S, Ahantharik P, Laohawiwattanakul S, Premsuk A, Ratanapo S: "Efficacy of fermented teas in antibacterial activity." Kasetsart J Nat Sci 2006;40:925–933.
[16] Mo H, Zhu Y, Chen Z: "Microbial fermented tea—a potential source of natural food preservatives."Trends Food Sci Technol 2008;19:124–130.
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